journal of clinical investigation insight impact factor

Qian Li, Xiaoning Han, Xi Lan, Yufeng Gao, Jieru Wan, Frederick Durham, Tian Cheng, Jie Yang, Zhongyu Wang, Chao Jiang, Mingyao Ying, Raymond C. Koehler, Brent R. Stockwell, Jian Wang. インパクトファクター は、自然科学・社会科学分野の学術雑誌を対象として、その雑誌の影響度、引用された頻度を測る指標である。インパクトファクター特定の年の平均引用数は、最初の2年間に発表された論文の総数から計算されました。たとえばある雑誌の2019年のインパクトファクターは2017年と2018年の論文数、2019年のその雑誌の被引用回数から次のように求める。 The chart shows the evolution of the average number of times documents published in a journal in the past two, three and four years have been cited in the current year. Several highly cited articles that contributed to the Impact Factor are listed below. 10.510 (2020年の最新データ)。 The set of journals have been ranked according to their SJR and divided into four equal groups, four quartiles. The findings recapitulate those of previous studies of common cancers but also identify alterations, including in AXL and EGFR, associated with response to targeted therapies. We utilized single-cell RNA sequencing (scRNA-seq) to identify epithelial cell types and associated biological processes involved in the pathogenesis of IPF. Data Source: Scopus®, Metrics based on Scopus® data as of April 2020, The American Society for Clinical Investigation. 学術雑誌査読速度、 インパクトファクタートレンド予測システムは、学術研究者が豊富なインテリジェンスと膨大な情報を通じて将来のジャーナルの影響とパフォーマンスを予測するのに役立つオープンで透明なプラットフォームを提供します。インパクトファクタートレンド予測システムは、追加の暗号化操作、隠れたファクター、またはシステムの遅延なしに、大量のデータを即座に提示します。, Journal of Clinical InvestigationのISSNは00219738です。ISSNは、さまざまな新聞、雑誌、雑誌、雑誌、および印刷媒体と電子媒体のすべてのメディアを識別するために使用される8桁のコードです。, Journal of Clinical Investigation - ジャーナルを購読する (non-OA), Journal of Clinical Investigationは購読ジャーナルです(non-OA)。出版社は、雑誌の記事の著作権を所有しています。 記事を読みたいと思う人はだれでもこれらの記事にアクセスする個人か代理店によってそれのために支払うべきである。これらの記事を何らかの方法で使用する場合は、出版社から許可を得る必要があります。, Journal of Clinical Investigationの発行元は American Society for Clinical Investigation The objective of this study was to investigate whether telomere dysfunction in type II AECs, mediated by deletion of the telomere shelterin protein TRF1, leads to pulmonary fibrosis in mice (SPC-Cre TRF1fl/fl mice). We found that administration of ferrostatin-1, a specific inhibitor of ferroptosis, prevented neuronal death and reduced iron deposition induced by hemoglobin in organotypic hippocampal slice cultures (OHSCs). Evolution of the number of total citation per document and external citation per document (i.e. Gene expression profiling revealed treatment-related downregulation of inflammatory markers, including signatures for CTLs and IFN response genes and upregulation of hair-specific markers. JCI Insight’s 2018 Impact Factor, the first for the journal, is 6.014. Idiopathic pulmonary fibrosis (IPF) is a lethal interstitial lung disease characterized by airway remodeling, inflammation, alveolar destruction, and fibrosis. The chart shows the evolution of the average number of times documents published in a journal in the past two, three and four years have been cited in the current year. The present study provides a rich data source to further explore lung health and disease. journal self-citations removed) received by a journal's published documents during the three previous years. This work was supported by DFCI, BWH, and the National Cancer Institute (5R33CA155554 and 5K23CA157631). When sufficient DNA was obtained, OncoPanel yielded a result in 96% of cases. CONCLUSION. JAMA - Journal of the American Medical Association, B=対象の雑誌が2018年に掲載した論文数; SPC-Cre TRF1fl/fl mice will be useful for assessing cellular and molecular mechanisms of lung fibrosis mediated by telomere dysfunction. Telomeres are short in type II alveolar epithelial cells (AECs) of patients with idiopathic pulmonary fibrosis (IPF). Authors may transfer research manuscripts from the preprint servers bioRxiv and medRxiv for submission to the JCI. Follow us on @ScimagoJRScimago Lab, Copyright 2007-2020. 25% scientists expect Journal of Clinical Investigation Journal Impact 2019-20 will be in the range of 14.5 ~ 15.0. People also search for: These results indicate that ferroptosis contributes to neuronal death after ICH, that administration of ferrostatin-1 protects hemorrhagic brain, and that cyclooxygenase-2 could be a biomarker of ferroptosis. C=対象の雑誌が2017年,2018年に掲載した論文が、2019年に引用された延べ回数; 学術雑誌人気のリサーチおよび CONCLUSIONS. Nine of twelve patients (75%) demonstrated a remarkable response to treatment, with average hair regrowth of 92% at the end of treatment. 学術雑誌テンプレートを提供し, 一連の関連ジャーナルの重要な指標には The two years line is equivalent to journal impact factor ™ (Thomson Reuters) metric. The journal publishes basic and phase I/II clinical research submissions in all biomedical specialties, including Autoimmunity, Gastroenterology, Immunology, Metabolism, Nephrology, Neuroscience, Oncology, Pulmonology, Vascular Biology, and many others. Nature Reviews Drug Discovery, Individual IPF cells frequently coexpressed alveolar type 1 (AT1), AT2, and conducting airway selective markers, demonstrating “indeterminate” states of differentiation not seen in normal lung development. Alopecia areata (AA) is a common autoimmune disease with a lifetime risk of 1.7%; there are no FDA-approved treatments for AA. JCI Insight’s 2018 Impact Factor, the first for the journal, is 6.014. In this study, we first demonstrated that ferroptosis, a newly identified form of cell death, occurs in the collagenase-induced ICH model in mice. Journal impact search engine. The Journal Impact 2019 of Journal of Clinical Investigation is A=対象の雑誌が2017年に掲載した論文数; Q1. Moreover, ferrostatin-1 in combination with other inhibitors that target different forms of cell death prevented hemoglobin-induced cell death in OHSCs and human induced pluripotent stem cell–derived neurons better than any inhibitor alone. We initiated an open-label clinical trial of 12 patients with moderate-to-severe AA, using oral ruxolitinib, 20 mg twice per day, for 3–6 months of treatment followed by 3 months follow-up off drug. scRNA-seq analyses identified loss of normal epithelial cell identities and unique contributions of epithelial cells to the pathogenesis of IPF. METHODS. Journal Impact Trend Forecasting System displays the exact community-driven Data without secret algorithms, hidden factors, or systematic delay. We deployed a hybrid-capture and massively parallel sequencing assay (OncoPanel) for all adult and pediatric patients at our combined cancer centers. 学術雑誌合格-拒絶 率、 In rare cancers, potentially actionable alterations suggest the utility of a “cancer-agnostic” approach in genomic profiling. Clinicaltrials.gov NCT01950780. The users of Scimago Journal & Country Rank have the possibility to dialogue through comments linked to a specific journal. Ratio of a journal's items, grouped in three years windows, that have been cited at least once vs. those not cited during the following year. Our cohort consists of cancer patients unrestricted by disease site or stage. Q1。 10.510, which is just updated in 2020. Yan Xu, Takako Mizuno, Anusha Sridharan, Yina Du, Minzhe Guo, Jie Tang, Kathryn A. Wikenheiser-Brokamp, Anne-Karina T. Perl, Vincent A. Funari, Jason J. Gokey, Barry R. Stripp, Jeffrey A. Whitsett. RESULTS. Self-Citation Ratio、 Additionally, we found that ferrostatin-1 reduced lipid reactive oxygen species production and attenuated the increased expression level of PTGS2 and its gene product cyclooxygenase-2 ex vivo and in vivo. H-Index、 We thank the journal’s community of authors, reviewers, editors, and readers who contributed to the journal’s success since its launch in 2016. Journal of Clinical Investigation - インパクトファクター. Medicine (miscellaneous) (Q1)。, Journal of Clinical Investigation - 重要な指標, 実際、単一の指標のみを使用してJournal of Clinical Investigationジャーナルの影響を完全に理解することは困難であるため、貢献への参照を提供するにはより多くの指標が必要です。学術アクセラレーターは、学術雑誌略称 ISO4、 In contrast, scRNA-seq of IPF cells identified 3 distinct subsets of epithelial cell types with characteristics of conducting airway basal and goblet cells and an additional atypical transitional cell that contributes to pathological processes in IPF. We conclude that telomere dysfunction in type II AECs is sufficient to cause lung fibrosis, and may be a dominant molecular defect causing IPF. Locks of Love Foundation, the Alopecia Areata Initiative, NIH/National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), and the Irving Institute for Clinical and Translational Research/Columbia University Medical Center Clinical and Translational Science Award (CUMC CTSA). Comprehensive genomic profiling of a patient’s cancer can be used to diagnose, monitor, and recommend treatment. Deletion of TRF1 in type II AECs for up to 9 months resulted in short telomeres and lung remodeling characterized by increased numbers of type II AECs, α-smooth muscle actin+ mesenchymal cells, collagen deposition, and accumulation of senescence-associated β-galactosidase+ lung epithelial cells. more. We previously identified a dominant IFN-γ transcriptional signature in cytotoxic T lymphocytes (CTLs) in human and mouse AA skin and showed that treatment with JAK inhibitors induced durable hair regrowth in mice by targeting this pathway. Deletion of TRF1 in collagen-expressing cells caused pulmonary edema, but not fibrosis. The Journal Impact Quartile of Journal of Clinical Investigation is The two years line is equivalent to journal impact factor ™ (Thomson Reuters) metric. In addition to the 2-year Journal Impact, the 3年インパクトファクター can provide further insights into the impact of Journal of Clinical Investigation. SNIP (Source Normalized Impact per Paper)が含まれます,Journal of Clinical Investigationジャーナルの影響をさらに評価できます。, American Society for Clinical Investigation, JAMA - Journal of the American Medical Association, SNIP (Source Normalized Impact per Paper). 減少しました。 Journal Impact Trend Forecasting System provides an open, transparent, and straightforward platform to help academic researchers Predict future journal impact and performance through the wisdom of crowds. Not every article in a journal is considered primary research and therefore "citable", this chart shows the ratio of a journal's articles including substantial research (research articles, conference papers and reviews) in three year windows vs. those documents other than research articles, reviews and conference papers. Compared with historical Journal Impact data, the Journal Impact 2018 of Journal of Clinical Investigation dropped by Developments in the clinical research lead to novel diagnostic procedures, drug design and development and innovative delivery mechanism. Nature Reviews Immunology, The purpose is to have a forum in which general doubts about the processes of publication in the journal, experiences and other issues derived from the publication of papers are resolved. Several highly cited articles that contributed to the Impact Factor are listed below. For topics on particular articles, maintain the dialogue through the usual channels with your editor. Deletion of TRF1 in type II AECs for 2 weeks increased γH2AX DNA damage foci, but not histopathologic changes in the lung. Genomic results may alter management in diverse scenarios; however, additional barriers must be overcome to enable precision cancer medicine on a large scale. 25% scientists expect Journal of Clinical Investigation Journal Impact 2019-20 will be in the range of 14.5 ~ 15.0. Evolution of the total number of citations and journal's self-citations received by a journal's published documents during the three previous years. ∴C÷(A+B)=2019年のインパクトファクター 。 11.08% Read more about the ASCI. The Journal Impact Factor should be used with informed peer review. It measures the scientific influence of the average article in a journal, it expresses how central to the global scientific discussion an average article of the journal is.

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